06-12-2016, 01:26 AM
(This post was last modified: 06-12-2016, 04:50 AM by VergeOfDiscovery.)
I had told a friend (in medical related field) at a party that DMAE & Centophenoxine have shown some promise in reducing/removing beta amyloid plaques...a chief suspected culprit when it comes to Alzheimer's.
While looking for references to that, I came across research (I'm sure I've seen this before, but taking another look at it in this (NBE) context).
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2390933/
pharmacological depletion of androgens and inhibition of androgen signaling in men may be predicted to yield deleterious cognitive consequences
and that androgen depletion – either through normal aging or pharmacological action – can result in specific cognitive impairments
What's interesting are the implications for intentionally trying to reduce androgens (and not the report's leaning towards promoting androgen therapy). Section 4 may be of interest in what that means in relation to estrogen.
While looking for references to that, I came across research (I'm sure I've seen this before, but taking another look at it in this (NBE) context).
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2390933/
pharmacological depletion of androgens and inhibition of androgen signaling in men may be predicted to yield deleterious cognitive consequences
and that androgen depletion – either through normal aging or pharmacological action – can result in specific cognitive impairments
What's interesting are the implications for intentionally trying to reduce androgens (and not the report's leaning towards promoting androgen therapy). Section 4 may be of interest in what that means in relation to estrogen.