20-04-2015, 09:30 PM
(17-04-2015, 08:57 AM)-Clelia- Wrote: well done Lotus, you found the answer at this question:
"This is a big deal, so anything over
3000 mg of arachidonic acid AA goes to PGE2, but this course (pathway) up regulate cancer cells (or said to be), what can stop that conversion process?"
Omega 3 fatty acids stop the cancer process.
So a good and healty program should include: omega 3-6 in a proper ratio (1:3, or 1:4), and GLA. Which amounts do you suggest?
Hi Clelia,
Good point about upping omega 3's, and thanks for the input, from what I've read there are certain omega 6's that create inflammation, GLA isn't one of them, e.g. EPO, borage oil, black currant oil. GLA is converted to DGLA which reduces inflammation. PGE2 concerns me, PGE1 reduces inflammation, although I'm not a fan of NSAIDS, (but I consider them to be pro-aromatase). EGF and PGE2 is a serious looking contender for aromatase potential.
IMO EPO is a EFA that should be in an NBE program, it takes consistent use (4-6 months) 2000 to 3000 mg (possibility to increase) before the full benefits are seen. But I see thats par for the course considering NBE is long term. But EFA's are something we should be using long term anyways.