25-02-2018, 04:16 AM
(This post was last modified: 25-02-2018, 04:24 AM by tanysquirrel.)
not "wishful thinking" .....
http://www.hormones.gr/693/article/sex-d...-sex….html
"A. Disorders of gonadal (ovarian) development
1. [i]Ovotesticular[/i] DSD, previously named true hermaphroditism. It is a very rare disorder defined by the presence of both ovarian and testicular tissue in the same individual (Figure 4). In infancy the gonads appear to have normal ovarian tissue with numerous follicles and normal testicular tissue with seminiferous tubules containing germ cells. However, as time passes the ovarian tissue usually becomes functional and the testicular tissue regresses, becoming dysgenetic and showing interstitial fibrosis and lack of spermogonia.42,43 Besides the presence of ovotestis, ovary and testis may be found separately.42 Ovarian tissue is most often found on the left side and testicular tissue on the right side.42 There may be a mixture of Müllerian and Wolffian derivatives; in most patients uterus or unicornus uterus is observed. The external genitalia are ambiguous with various degrees of virilization, while, rarely, normal female genitalia may be present.44,45 The SRY gene (located on chromosome Yp11.3, OMIM#480000) in 46,XX ovotesticular patients is present in approximately 1/3 of the cases.43,44 In the SRY negative cases the mechanism responsible for the presence of testicular tissue is unclear. It may be that some patients have Y sequences confined to testicular tissue (therefore escaping detection via analysis of leucocyte DNA), there may be autosomal or X linked mutations in unknown genes controlling testicular development or there may have been an early presence of SRY sufficient to differentiate testicular tissue but vanishing later on.43,46 A mosaicism with a Y bearing cell line in the gonad is another possibility, as the SRY gene is expressed in the ovotestes of XX ovotesticular DSD.47
Perhaps you mistake my logging of a weird side effect as a fanciful and delusional desire. Anyone who knows me, (on here and otherwise), knows I am of sound mind, am scientifically minded, and far from "crazy". Of all the posts I have made. 99% of them are sound, with research behind them. This is not meant for arguments sake, just for clarification.
Of course, I do not believe I fall under the above listed syndrome, however, I would need Chromozonal testing, imaging results, etc. All of which I do not think is necessary to prove a point. I know how I was born, I know where I am going. The above article was provided to show that there is much more than the standard XX /XY variation. Nature can be a funny thing.
http://www.hormones.gr/693/article/sex-d...-sex….html
"A. Disorders of gonadal (ovarian) development
1. [i]Ovotesticular[/i] DSD, previously named true hermaphroditism. It is a very rare disorder defined by the presence of both ovarian and testicular tissue in the same individual (Figure 4). In infancy the gonads appear to have normal ovarian tissue with numerous follicles and normal testicular tissue with seminiferous tubules containing germ cells. However, as time passes the ovarian tissue usually becomes functional and the testicular tissue regresses, becoming dysgenetic and showing interstitial fibrosis and lack of spermogonia.42,43 Besides the presence of ovotestis, ovary and testis may be found separately.42 Ovarian tissue is most often found on the left side and testicular tissue on the right side.42 There may be a mixture of Müllerian and Wolffian derivatives; in most patients uterus or unicornus uterus is observed. The external genitalia are ambiguous with various degrees of virilization, while, rarely, normal female genitalia may be present.44,45 The SRY gene (located on chromosome Yp11.3, OMIM#480000) in 46,XX ovotesticular patients is present in approximately 1/3 of the cases.43,44 In the SRY negative cases the mechanism responsible for the presence of testicular tissue is unclear. It may be that some patients have Y sequences confined to testicular tissue (therefore escaping detection via analysis of leucocyte DNA), there may be autosomal or X linked mutations in unknown genes controlling testicular development or there may have been an early presence of SRY sufficient to differentiate testicular tissue but vanishing later on.43,46 A mosaicism with a Y bearing cell line in the gonad is another possibility, as the SRY gene is expressed in the ovotestes of XX ovotesticular DSD.47
Perhaps you mistake my logging of a weird side effect as a fanciful and delusional desire. Anyone who knows me, (on here and otherwise), knows I am of sound mind, am scientifically minded, and far from "crazy". Of all the posts I have made. 99% of them are sound, with research behind them. This is not meant for arguments sake, just for clarification.
Of course, I do not believe I fall under the above listed syndrome, however, I would need Chromozonal testing, imaging results, etc. All of which I do not think is necessary to prove a point. I know how I was born, I know where I am going. The above article was provided to show that there is much more than the standard XX /XY variation. Nature can be a funny thing.