[Luannep]

What the difference if I used White Peony herbal tea instead of extract?

[Lenneth]

extract is far more potent.

[AbiDrew85]

What the difference if I used White Peony herbal tea instead of extract?

extract is far more potent.

Actually, most "white peony" "herbal" tea isn't even actually white peony, so there's that too. If it specifically mentions the plant, peoniflorin lactifloria iirc, then it's white peony, if it doesn't... it may be something else!

That said... White peony is worthless to people without ovaries. You want Green Tea Extract, specifically, EGCG

[Luannep]

What the difference if I used White Peony herbal tea instead of extract?

extract is far more potent.

Actually, most "white peony" "herbal" tea isn't even actually white peony, so there's that too. If it specifically mentions the plant, peoniflorin lactifloria iirc, then it's white peony, if it doesn't... it may be something else!

That said... White peony is worthless to people without ovaries. You want Green Tea Extract, specifically, EGCG

Thank You

[Marcy]

That said... White peony is worthless to people without ovaries. You want Green Tea Extract, specifically, EGCG

I thought white peony was a good aromatase promoter and blocks 5ar?

[jannet.duff]

That said... White peony is worthless to people without ovaries. You want Green Tea Extract, specifically, EGCG

I thought white peony was a good aromatase promoter and blocks 5ar?

I suspect that comment is towards tea, not extract. I know WP extract makes a big difference for me.

[Lotus]

Don't know if this will help but I just read on another thread that WP will do nothing for someone with no ovaries (said by Abi Drew I believe). Not sure though. They suggested the gte with ecgc's as well.

You don't need just ovaries to promote aromatase, a liver, fat cells, bone, brain, germ cells, etc are potential targets of aromatase.

White peony (Shakuyaku-Kanzo-To) promotes the activity of aromatase from the synthesis of estradiol from testosterone, which in turn lowers serum T levels.

It's worth repeating, as long as you produce aromatase, using a " pro-aromatase " (e.g. White Peony) will promote its synthesis. WP has benefited many here at BN pursuing NBE (for both male and female alike, self and 4+ cups included). White Peony is not the only pro-aromatase available, EPO (evening primrose oil, borage oil, forskolin, vitamin D, horny goat weed [ICARIN], green tea, second messengers, Genistien, PGE2, COX2, Leptin [a biggie]), others too.


But.........

If we really want to blow the doors off aromatase we'd turn our attention to EGFR(epiderrmal growth factor), which sits of the outside of cells, and is essential part Mammary Gland Development. So, the question is " how come we don't know about it? ", my guess?.....its too F**kin complicated lol. I'll take at stab at it though: please pay note to the highlighted areas, I believe EGFR combined and inhibiting histone deacetylation (HDAC) while upregulating PGE2, should make for the strongest aromatase.......say what?, broad strokes of course, I'm still working on the rough edges as to how it translates to NBE. over stimulus concerns are top priority. In other words, if we can find away around the trouble spots, it lessens proliferation.


function of the epidermal growth factor receptor and erbB-2 during mammary gland morphogenesis.
Sebastian J1, Richards RG, Walker MP, Wiesen JF, Werb Z, Derynck R, Hom YK, Cunha GR, DiAugustine RP.
Author information
Abstract
The hormonal stimulation of mammary gland morphogenesis is believed to occur through growth factor receptor signaling pathways. To determine the importance of the epidermal growth factor receptor (EGFR) pathway, we examined extracts of inguinal mammary glands from prepubertal and pubertal mice for tyrosine-phosphorylated EGFR and other erbB receptors. Tyrosine phosphorylation of both EGFR and erbB-2 was detected in normal female BALB/c mice at 5-6 weeks of age, but not during the prepubertal stage, e.g., 24 days of age. Treatment of mice with estradiol or epidermal growth factor also stimulated the formation of mammary EGFR/erbB-2 phosphotyrosine. Waved-2 mice, which have impaired EGFR kinase activity, exhibited less mammary development than did wild-type (wt) mice when both were evaluated at 36 days of age. Because EGFR knockout (KO) mice die shortly after birth, glands from the newborns were implanted under the renal capsules of female nude mice. Under these conditions, extensive ductal growth was observed in mammary glands from wt animals; in contrast, glands from EGFR KO mice failed to grow beyond rudimentary structures. Tissue recombinants revealed that the wt fat pad supported the morphogenesis of EGFR KO epithelium, whereas the EGFR KO fat pad did not. Taken together, these data suggest that EGFR is essential for morphogenesis of the mammary ducts and functions during this period of mammary development as a heterodimer with erbB-2 in the mammary stroma.






http://www.intechopen.com/books/malignan...esotheliom

[VergeOfDiscovery]

Re: most "white peony" "herbal" tea isn't even actually white peony

I was at a Vitamin Shoppe here today and although they had several teas marked as White Peony, none of them were White Peony!

The only actual white peony product they sell (from Life Extension) they did not have.

But Swanson has a two for one on their own today at almost 1/3 the price (although full spectrum).