[myboobs]

Remember in the first post of the thread this statement:


Men produce 3mg to 10mg of T per day, and of that 4% gets converted to DHT and .02% gets converted to estrogen, (estradiol E2)


The prostate converts 95% of T to DHT by 5 alpha reductase, ( 5ar).


.02% of E is converted by the enzyme aromatase, this small percentage can be misleading, estradiol is 100 more potent at the receptor sites then T, that means T needs to balance at the same level to have the same affinity.



Although naturally-occurring estrogens circulate in the blood largely bound to sex hormone-binding globulin and albumin, only unbound estrogens enter target tissue cells. This statement is explaining Free E, exactly the same principle as FREE T and that also means E-receptors.

Below is what I'm talking about if anybody would like some supported info:

Testosterone-derived estradiol production by male endothelium is robust and dependent on p450 aromatase via estrogen receptor alpha.
http://www.springerplus.com/content/2/1/214

Let me throw a spanner in the works .
Since it is the pituitary gland that sends signals for production of various hormones and also signals for balancing any unbalances .

I was watching a health program where this guy's breast ( albeit small ) was producing milk , he was diagnosed by the doctor pituitary gland malfunction for which this guy had treatment .

So here is me thinking maybe we at NBE could look at manipulating the pituitary gland . As by doing that you are tackling the source rather than end product.

Is my logic correct ?