That is interesting.

Theoretically if there are 2 units of free T. One unit of T is converted into (not necessarily 1 unit of ) Estrogen. The free T that works against progress is negligible, Estrogen:Testosterone 100 to 1 (when you say receptor sites, different receptor sites respond differently) . (As long as the 1 unit of testosterone doesn't become less than 1/100 unit of estrogen) So free T is also necessary for overall function. Free T Might also up-regulate Estrogen receptors. From a study, it said testosterone was negatively Correlated (correlation is not causation) with growth during luteal phase. This sentence is speculation, perhaps it was DHT, or not free T. It could have also been free T, or its indirect effects on DHT or estrogen. Everyone seems to agree that DHT is more inhibiitive than free T. This is a good argument for aromatase, it MIGHT convert free T into estrogen Instead of DHT.

So, I don't know if DHT has an essential function for females. DHT must have a function for males, but the more there is, the more unwanted effects it has for both genders.

I've come across papers that mentioned DH versions of progestogens. I have no idea what they do.