02-05-2015, 05:57 AM
(02-05-2015, 05:19 AM)froger Wrote: What I would want to know is how to limit DHT enough to grow breasts but not enough to lose erections. I know I don't have the answer because so far erections haven't stopped or even slowed for me in the two years since I started. I have had growth and some different fat allocation (hate to say redistribution), but not as much as I think I can achieve. I think I ideally if you can reduce DHT, leave some remain to do what it does, then backdoor convert the rest you may come close to the best of both worlds.
Froger,
Option #1
I think we could walk the line with something like Finasteride and 25mg to 50mg of DHEA, androstenedione and DHEA are pro-aromatase. If estradiol is higher taking DHEA will go to aromatase, and whatever T is left over by Finsateride still on the table could get the lobido going. It's possible this action could be applied by reishi?, or using something like EFA's (essential fatty acids) epo or borage, which is about 85% ant DHT.
Option #2
Spiro- Although I'm less certain about this approach, meaning:
*Spiro displaces estrogen from SHBG, which lowers free estrogens
*Blocks tetosterones synthesis
*Binds to estrogen receptors (don't know which one yet)
option #3
cAMP or cyclic adenosine monophosphate is another possible NBE tool. Imo cAMP ties into fatty acids, or in large part how cAMP delivers FA's to hormones. In other words increasing blood flow to the receptors increase their bioavailability. Forilskin comes to mind, but quite honestly I think fatty acids work quite the same way.
cAMP is a second messenger important in many biological processes. cAMP is derived from adenosine triphosphate (ATP) and used for intracellular signal transduction in many different organisms, conveying the cAMP-dependent pathway.
Yes it's the same response seen from Forskolin-, but I believe there's other options besides forskolin though, which good luck finding the real thing.......to be continued (searching)

