[Lotus]

Wow, Lotus, I'll try to study what you wrote in above threads and then apply them with fasting. I hope HIIT after fasting will not shrink me, I don't weight a lot for a bio male- Thanks again, POM

I understand, let me explain it like this, if your baseline T levels are already low pre-work out, and you supplement with anti-androgens it should stand to reason you'll still see a slight bump in T. But, according to the study below fatty acids did not increase cortisol, which, increased cortisol drives T levels up, (e.g. stress). In order to grow breasts we still need T, which T converts to E through aromatase, (that part is indisputable). Honestly, (IMO) I don't see it as a problem. Take a look at this study below, (see the note at the bottom, I'll provide other studies regarding HIIT).


High-intensity training produces free radicals, but it triggers an abundance of repair peptides, enzymes and hormones to be released. The net effect of this is healthy tissue repair and favorable effects on body composition and anti-aging qualities.

Testosterone and cortisol in relationship to dietary nutrients and resistance exercise.
Volek JS1, Kraemer WJ, Bush JA, Incledon T, Boetes M.
Author information
Abstract
Manipulation of resistance exercise variables (i.e., intensity, volume, and rest periods) affects the endocrine response to exercise; however, the influence of dietary nutrients on basal and exercise-induced concentrations of hormones is less understood. The present study examined the relationship between dietary nutrients and resting and exercise-induced blood concentrations of testosterone (T) and cortisol ©. Twelve men performed a bench press exercise protocol (5 sets to failure using a 10-repetitions maximum load) and a jump squat protocol (5 sets of 10 repetitions using 30% of each subject's 1-repetition maximum squat) with 2 min of rest between all sets. A blood sample was obtained at preexercise and 5 min postexercise for determination of serum T and C. Subjects also completed detailed dietary food records for a total of 17 days. There was a significant (P < or = 0.05) increase in postexercise T compared with preexercise values for both the bench press (7.4%) and jump squat (15.1%) protocols; however, C was not significantly different from preexercise concentrations. Significant correlations were observed between preexercise T and percent energy protein (r = -0.71), percent energy fat (r = 0.72), saturated fatty acids (g.1,000 kcal-1.day-1; r = 0.77), monounsaturated fatty acids (g.1,000 kcal-1.day-1; r = 0.79, the polyunsaturated fat-to-saturated fat ratio (r = -0.63), and the protein-to-carbohydrate ratio (r = -0.59). There were no significant correlations observed between any nutritional variables and preexercise C or the absolute increase in T and C after exercise. These data confirm that high-intensity resistance exercise results in elevated postexercise T concentrations. A more impressive finding was that dietary nutrients may be capable of modulating resting concentrations of T.

The bigger thing is the growth hormone increase, which is what we're after.