24-07-2016, 08:33 PM
(24-07-2016, 12:05 AM)iaboy Wrote: O.k., Here is mine from 2 weeks ago. Still confused as how I haven't done any herbs ect for about 1 month now, and no discernible loss of breast fat or tissue.
I hope this will help you
Testost SerPl-msCnc 608.10 Testosterone, Total TIC ( I would of thought this would of been around 300, myself)
Lutenizing Hormone 14.15 ( Post Menopausal ??)
Estadiol 38.00 Pg/nl (Too low, for what I have accomplished?)
Beta HCG Quant <2.00 (Not sure why this was taken, cause I think it's for pregnancy??? LOL)
Albumin 4.5 g/dl ( Results from 4/15/16 )
Maybe their gauging negative feedback control of gonadotropin releasing hormone (GnRH) by the hypothalamus. I read that " continuous GnRH stimulation of gonadotrope cells in the pituitary increases LH and FSH production as well as pituitary hCG. " (just a guess)

Here's a personal observation based on seeing some lab tests (and not limited to this thread only). What if what we're seeing is that total T has no bearing?, meaning total T isn't active. The true bio-active hormones are not bound too SHBG, (meaning tied up).
And the way we can tell is from the free T percentage and bioactive T (testosterone) . Here's what I mean, my total T is almost 0 (3 ng/dL) and yet my free T percentage (the active amount of androgen, albeit free T) is the same as everyone else, and pretty much my bioT percentage too (being the same). And the other tell is the wild card SHBG, it looks like PM/nbe pushes SHBG up in genetic males.
My SHBG came back lower to 52, from 77 nmol/L........and with the decrease it showed a higher percentage of free T. Now, if I don't capitalize on this free T increase it'll go to DHT. This is scientifically proven fact about how DHT preferentially binds to SHBG.
If we can't control the rate of DHT binding, perhaps we merely render the metabolism of DHT inactive. This option is compilcated to explain, you see it's using 17beta HSD (17Beta Hydroxysteroid dehydrogenase), picture it as a process of completely shutting DHT out of the cell receptor, like " red alert........shields up " denying access.
Thanks for the results iaboy. I think your in the same boat as Sofia, spanky, Marcy and others, meaning PM, and other nbe parts raise total T but keep free T low giving way to more phytoestrogens to block DHT,--------the issue of not seeing net estrogens showing up in blood is perplexing though, but in the back of our minds I think we knew this already (PM not showing up in the blood) though not expressed technically.

