31-08-2016, 11:57 PM
Using progesterone to offset estrogen and/or E-dominance is one thing, the example of using progesterone in combination with E2 resulted in more breast tissue in the MTF study participants.
In males, the amount of estrogen and progesterone receptors are close to being the same, followed by androgen and glucocorticoid receptors. Personally, I don't think we need much PC, maybe about 1/8 teaspoon (about a fingertip).
When combination hormone therapy is used, multiple studies prove there's more growth (posted studies @ BN), a synthetic example of this is the combination birth control pill......E2/progestin, (I wouldn't use pharma progestins personally). Whether you choose to use on PC on the breasts, inner wrists, or rectal applications is up to the user.
In a pubertal surge most all hormones are released, it as after all what we're trying to mirror (the surge). Natural substitute of progesterone would be black walnut, and my favorite natural substitution for progesterone is " avocados ".
The other important thing progesterone does for NBE is the reset of receptor sensitivity, and as we know too much estrogen desensitizes estrogen receptors.
In males, the amount of estrogen and progesterone receptors are close to being the same, followed by androgen and glucocorticoid receptors. Personally, I don't think we need much PC, maybe about 1/8 teaspoon (about a fingertip).
When combination hormone therapy is used, multiple studies prove there's more growth (posted studies @ BN), a synthetic example of this is the combination birth control pill......E2/progestin, (I wouldn't use pharma progestins personally). Whether you choose to use on PC on the breasts, inner wrists, or rectal applications is up to the user.
In a pubertal surge most all hormones are released, it as after all what we're trying to mirror (the surge). Natural substitute of progesterone would be black walnut, and my favorite natural substitution for progesterone is " avocados ".
The other important thing progesterone does for NBE is the reset of receptor sensitivity, and as we know too much estrogen desensitizes estrogen receptors.