I stopped taking SP a few months ago, on the reasoning that, if my T level was near zero, my DHT level must also be near zero, and therefore I didn’t need it. Recently I was reminded that SP was an NBE herb for women long before it began to be taken by men for BPH. This led me to some further research into SP, from which I am forced to the conclusion that no one really knows for certain how SP does what it does, assuming that it does it. Just a couple of links illustrative of this are:
www.aaronsworld.com/Bastyr/Class%20Notes/Bot%20Med/Bot%20med%20III/Serenoa_repens.pdf
and
www.drugs.com/npp/saw-palmetto.html
The diagram on page 7 of the first link I find particularly interesting. Since it appears not to be a significant source of phytoestrogens, or anything likely to interfere significantly with PM, my query is whether there is likely to be any benefit to my taking it (I have about 3 months supply of 320mg extract capsules so there is no extra expense)? If one of its properties is to assist in the utilisation of estrogen does this also apply to phytoestrogens? Is some of the effectivenes of SP in alleviating LUTS due to an antimuscarinic effect reducing overactive bladder symptoms?
www.aaronsworld.com/Bastyr/Class%20Notes/Bot%20Med/Bot%20med%20III/Serenoa_repens.pdf
and
www.drugs.com/npp/saw-palmetto.html
The diagram on page 7 of the first link I find particularly interesting. Since it appears not to be a significant source of phytoestrogens, or anything likely to interfere significantly with PM, my query is whether there is likely to be any benefit to my taking it (I have about 3 months supply of 320mg extract capsules so there is no extra expense)? If one of its properties is to assist in the utilisation of estrogen does this also apply to phytoestrogens? Is some of the effectivenes of SP in alleviating LUTS due to an antimuscarinic effect reducing overactive bladder symptoms?