[Lotus]

Thanks for the info; but what I mean is when you say "cycle" do you mean cycle between herbs or cycle on/off periods.

Ok, I understand now (thanks), yes, cycle on/off NBE herbs. The plasma levels of herbs most likely have shorter half lives than pharma meds, though who knows for sure really because of changes in batch controls (quality).

The build up of toxins from the multiple ingredients in herbals needs to be filtered out of the body (clearance), we owe ourselves at least that opportunity.

[Lotus]

I think it's like pretty much anything that you do in that you build up a tolerance if you are continuous without a break.

Lotus- on the capsules being only 20% absorption rate, what do you recommend to do with capsules if anything to get a better transfer and at what estimated rate?

Thanks

Hi peekaboobs,

I prefer extracts over capsules tbh, though I'd still have a smaller ratio of oral supps to extracts simply because some supplements need the liver metabolism. Sublingual misses most of the first pass metabolism. Transdermal delivery would be another favorable option, injections (pharma route) and rectal delivery are other options too.

[Stevenator.]

I'm curious about the oils listed for NBE. Are these pill form, or topical treatments. Now that winter is soon upon us, my skin is getting dry from shaving & I am wondering if any particular skin lotion/oil is recommended for NBE ?

Also, and this is perplexing ... NBE Foods. All the foods listed are just normal foods, and even with my breaks, I've continued to eat only NBE Focused Foods, but, what is the best way to take advantage of the properties they hold ??

Thank you.

[BeautifulBambi]

I just started using White Peony about how long do you think it should take to start feeling and seeing results from it?

[bunnysdead]

I just started using White Peony about how long do you think it should take to start feeling and seeing results from it?

Hi BeautifulBambi - I kind of measure the effectiveness of my herbal program against morning erections.  After a couple/few/week on supplements I notice my morning erections are either gone or weaker than normal.

[BlueCoral]

For licorice, should I be using licorice root or DGL?

Long time no see btw, I like the new layout of the site.

[Lotus]

For licorice, should I be using licorice root or DGL?

Long time no see btw, I like the new layout of the site.

hi BlueCoral, (I was thinking of you last week),  

dgl licorice removes most of the risks associated w/standard licorice root. However, I don't find either to be beneficial for NBE anymore. I say this based on the large amount of research I've found over the past 5 years (in my opinion fwiw).

Licorice root is counter productive for NBE/Hrt, dgl form too. 

LR oxidizes NAPDH and inhibits P450 enzymes,  what's more interesting (imo) is the latter part. Meaning I see LR inhibiting aromatase, not potentiating it. Oh sure LR binds to estrogen receptors, but that's where I think it ends. My reasoning is what it isn't doing to the P450 enzyme, (i.e. not synthesizing it). 

Agonistic and antagonistic estrogens in licorice root (Glycyrrhiza glabra)


Several fractions displayed higher responses than the maximum response obtained with the reference compound, the natural hormone 17β-estradiol (E2).


The roots of licorice are a rich source of flavonoids, in particular, prenylated flavonoids, such as the isoflavan glabridin and the isoflavene glabrene. Fractionation of an ethyl acetate extract from licorice root by centrifugal partitioning chromatography yielded 51 fractions. 
One third of the fractions displayed estrogenic activity towards either one or both estrogen receptors (ERs; ERα and ERβ). Glabrene-rich fractions displayed an estrogenic response, predominantly to the ERα. Surprisingly, glabridin did not exert agonistic activity to both ER subtypes. Several fractions displayed higher responses than the maximum response obtained with the reference compound, the natural hormone 17β-estradiol (E2). The estrogenic activities of all fractions, including this so-called superinduction, were clearly ER-mediated, as the estrogenic response was inhibited by 20–60% by known ER antagonists. Most fractions displaying superinduction were rich in flavonoids with single prenylation. Glabridin displayed ERα-selective antagonism, similar to the ERα-selective antagonist RU 58668. Whereas glabridin was able to reduce the estrogenic response of E2 by approximately 80% at 6 × 10−6 M, glabrene-rich fractions only exhibited agonistic responses, preferentially on ERα.
http://www.ncbi.nlm.nih.gov/pmc/articles...po=7.50000

[BlueCoral]

For licorice, should I be using licorice root or DGL?

Long time no see btw, I like the new layout of the site.

hi BlueCoral, (I was thinking of you last week),  

dgl licorice removes most of the risks associated w/standard licorice root. However, I don't find either to be beneficial for NBE anymore. I say this based on the large amount of research I've found over the past 5 years (in my opinion fwiw).

Licorice root is counter productive for NBE/Hrt, dgl form too. 

LR oxidizes NAPDH and inhibits P450 enzymes,  what's more interesting (imo) is the latter part. Meaning I see LR inhibiting aromatase, not potentiating it. Oh sure LR binds to estrogen receptors, but that's where I think it ends. My reasoning is what it isn't doing to the P450 enzyme, (i.e. not synthesizing it). 

Agonistic and antagonistic estrogens in licorice root (Glycyrrhiza glabra)


Several fractions displayed higher responses than the maximum response obtained with the reference compound, the natural hormone 17β-estradiol (E2).


The roots of licorice are a rich source of flavonoids, in particular, prenylated flavonoids, such as the isoflavan glabridin and the isoflavene glabrene. Fractionation of an ethyl acetate extract from licorice root by centrifugal partitioning chromatography yielded 51 fractions. 
One third of the fractions displayed estrogenic activity towards either one or both estrogen receptors (ERs; ERα and ERβ). Glabrene-rich fractions displayed an estrogenic response, predominantly to the ERα. Surprisingly, glabridin did not exert agonistic activity to both ER subtypes. Several fractions displayed higher responses than the maximum response obtained with the reference compound, the natural hormone 17β-estradiol (E2). The estrogenic activities of all fractions, including this so-called superinduction, were clearly ER-mediated, as the estrogenic response was inhibited by 20–60% by known ER antagonists. Most fractions displaying superinduction were rich in flavonoids with single prenylation. Glabridin displayed ERα-selective antagonism, similar to the ERα-selective antagonist RU 58668. Whereas glabridin was able to reduce the estrogenic response of E2 by approximately 80% at 6 × 10−6 M, glabrene-rich fractions only exhibited agonistic responses, preferentially on ERα.
http://www.ncbi.nlm.nih.gov/pmc/articles...po=7.50000

Good to see you to Lotus. 

I'm trying to figure out what to take, so that I don't take a herb that's doing the same thing as another.
How good is HGH vs. Gaba or Deer Velvet? Paba versus grape fruit?
Another test I'm thinking of is switching to Bovine Ovary (is it true that you need to maintain a certain body temp for it to work?) for estrogen, pro-aromatase, and 5-AR inhibiting; and Spearmint/Pygeum for an anti-androgen . Now that I'm thinking about starting again after a year, I think I'll not take PM since I've heard there's a global shortage of it recently or something.

[shaneny]

I had a quick question, if I'm taking FG, do I need something like SP for it to have any sort of actual affect? I also had briefly tried it with BC, but then read your posts that black cohosh is not good to take, so stopped. I'm guessing that was a wise move.

[Billie]

As we grow when is it suggested we start wearing a bra during the day or at night?