(09-09-2017, 01:52 PM)Tanya Marie Squirrel Wrote: (07-09-2017, 01:42 AM)Lotus Wrote: (03-09-2017, 09:33 AM)Tanya Marie Squirrel Wrote: I just ordered some royal jelly/vitamin E "pearls' from amazon...
https://www.amazon.com/gp/product/B000P00P7S/ref=oh_aui_detailpage_o00_s00?ie=UTF8&psc=1
if this works, as it seems to have on you.. then wow.. cant beat that at $6/bottle.....
I am sure "Individual miles may vary " aka it wont work for everyone, but if it does.. holy cow.
I have been in search to make my areolas/nips a bit larger than they are now (about half dollar in size for areola). I want that no doubt they are female areola size and larger nips (nips about .25" x.25")
Hi Squirrel,
RJ is uniquely amazing. Perhaps (just an observation) RJ is more effective at exploding areolas if you already have something to start with. RJ tip: warm RJ before application, it spreads easier...
How long before it really started changing the size of the areola? just a round about time would be nice
if NBE (or even hrt) hasn't changed the size or shape of one's areolas something is pevepreventing the process. I believe the simple truth is the T:E ratio (in short, not enough E). For me areola change (and growth) came from a combimation of experimenation...like red clover with PM, or PC plus PM. However, E2 (oral estradiol) combined with other things (like RJ) gives flexibility to areola growth (in my opinion). I'd give RJ at least 30 days, results my vary, in my case I saw changes fast (2-days).
There's another option I'm also excited about for areola growth, and that's E2 (oral estradiol) in combination with collagen/vitamin C supplement...and that combination is a big surprise. I take 2 mg E2 with 2000 mg Collagen w/vitamin C on an empty stomach, works quite effectively.
I'm also studying what CBD (cannabidiol) will do for breast growth, and the NBE science looks very promising, I'll put up the important highlights of adding CBD to NBE, and/or what it should do for breast growth.
Cannabidiol-induced intracellular Ca2+ elevations in hippocampal cells.
Drysdale AJ1, Ryan D, Pertwee RG, Platt B.
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Abstract
The phytocannabinoid cannabidiol (CBD) is at the forefront of therapeutic cannabinoid research due to its non-psychotropic properties. Research supports its use in a variety of disorders, yet the cellular mechanisms of its action remain unclear. In this study, the effect of CBD upon Ca2+ homeostasis in hippocampal cells was characterised. CBD (1 microM) elevated intracellular Ca2+ ([Ca2+]i) by approximately +45% of basal Ca2+ levels in both glia (77% responders) and neurones (51% responders). Responses to CBD were reduced in high excitability HEPES buffered solution (HBS), but not affected in low excitability/low Ca2+ HBS. CBD responses were also significantly reduced (by 50%) by the universal Ca2+ channel blocker cadmium (50 microM) and the L-type specific Ca2+ channel blocker nifedipine (20 microM). Interestingly, intracellular store depletion with thapsigargin (2 microM) had the most dramatic effect on CBD responses, leading on average to a full block of the response. Elevated CBD-induced [Ca2+]i responses (>+100%) were observed in the presence of the CB1 receptor antagonist, AM281 (1 microM), and the vanilloid receptor antagonist, capsazepine (CPZ, 1 microM). Overall, our data suggest that CBD modulates hippocampal [Ca2+]i homeostasis via intracellular Ca2+ stores and L-type VGCC-mediated Ca2+ entry, with tonic cannabinoid and vanilloid receptor signalling being negatively coupled to this pathway.